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However, under TNFR1 knockdown conditions, the expression levels of Rta and Zta in TNF--treated groups showed no significant reduction compared to positive controls, demonstrating that TNF- lost its suppressive effect on EBV reactivation when TNF-/TNFR1 signaling was compromised(Fig

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(b) In the combined case and control cohort of samples from the CB, aconitase activity was signicantly associated with GSH/GSSG (P0.01)

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